{"id":19910,"date":"2024-08-19T10:49:36","date_gmt":"2024-08-19T15:49:36","guid":{"rendered":"https:\/\/turnmedical.com\/?p=19910"},"modified":"2024-08-23T22:16:44","modified_gmt":"2024-08-24T03:16:44","slug":"etiology-acute-respiratory-distress-syndrome","status":"publish","type":"post","link":"https:\/\/turnmedical.com\/etiology-acute-respiratory-distress-syndrome\/","title":{"rendered":"Underlying Etiology of Acute Respiratory Distress Syndrome"},"content":{"rendered":"\t\t
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By Allison Varnum<\/span><\/em><\/p> In our previous blog, we defined acute respiratory distress syndrome (ARDS) and briefly discussed treatment options which include supportive measures and salvage therapies. To access that blog post, click here<\/a><\/strong>. In this blog, we will delve into the etiology of ARDS.<\/span><\/p> In ARDS, inflammatory cells bombard the lungs destroying the endothelial and epithelial borders causing the alveoli to be flooded with inflammatory cells, fluid, protein, and red blood cells. This injury can be intra- or extrapulmonary. Intrapulmonary, or direct, risk factors of ARDS are pneumonia, aspiration, pulmonary contusion, oxygen toxicity, near drowning, and inhalation injury. Extrapulmonary, or indirect, risk factors include sepsis, severe trauma, pancreatitis, multiple transfusions, overdose, and cardiopulmonary bypass1<\/span><\/span>. This cascade results in massive alveolar edema and begins the exudative phase2<\/span><\/span>.(A)<\/span><\/p>